By P. D. Evans
Insect body structure is at the moment present process a revolution with the elevated software of molecular organic options to enquire the molecular mechanisms underlying the physiological responses to insect cells.Advances in Insect body structure is commited to publishing prime quality studies on molecular biology and molecular genetics in components the place they supply an elevated knowing of physiological approaches in bugs. quantity 26 of this vintage seriescontinues to supply up to date experiences on topical matters of significance to all invertebrate physiologists and neurobiologists and comprises elevated assurance at the molecular biology of insect body structure.
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Additional info for Advances in Insect Physiology, Vol. 26
3) and some internal similarity to Torpedo acetylcholinesterase and bovine thyroglobulin, but its function is unknown. Unlike the Jhp21 protein, the 19K protein is not sex-specific. , 1988). Thus, the 19K protein, like the persistent storage protein, is produced constitutively but enhanced by JH. 3 Cold-hardiness and desiccation proteins. Different insects make use of several mechanisms to avoid the potentially lethal effects of extreme winter cold (Zachariassen, 1985). These include lowering the supercooling point by production of antifreeze proteins (as well as low molecular weight solutes such as polyols) and, in some instances, removal of lipoprotein ice nucleators from the haemolymph; in both processes JH appears to be involved.
In the same system, 20HE affects different proteins. However, in those cases in which the glands have not had previous exposure to JH, the action of J H appears to be biphasic. The best example is provided by the control of the synthesis of LHPl in Melanoplus. When long hyaline glands were removed from normal males and incubated in vitro with JH 111, the incorporation of [3H]-leucineinto LHPl occurred in a dose dependent fashion over a range from 40 to 60nM, with the response falling off at higher concentrations.
1983), and transplanting vitellogenic oocytes to JH-deficient hosts caused the oocytes to degenerate (Wilson, 1982). These observations confirm that JH is required throughout vitellogenesis. Diapausing Drosophila exhibit low titres of JH, and, although YP is present in the haemolymph, it fails to enter the follicles. , 1990). , 1981), JH increases the uptake of Vg into isolated ovarioles, and in Rhodnius (Davey and Huebner, 1974), incubation of isolated ovaries with Vg in vitro results in uptake of Vg only if JH is present.
Advances in Insect Physiology, Vol. 26 by P. D. Evans